A Review On The Viral Polymerase Inhibitor 7 Deaza 2

Throughout the history of humanity, different types of killer diseases occurred. Despite the severity of these diseases people always sought ways to alleviating them. Many of these epidemic diseases occurred as a result of pollution or changes in the environment. Recently, there have been the emergence of the most dangerous and killer diseases, many of them are infectious through different ways. Some of the most popular include HIV/AIDS, Ebola among others. However, while researchers and governments were struggling to alleviate and fight against HIV/AIDS there emerged a new form of viral disease that terrified the entire population of the planet. ZIKA virus is an emerging killer disease that has symptoms similar to a very mild dengue fever. This paper therefore is a review of an article entitled â€Å"The Viral Polymerase Inhibitor 7-Deaza-2’-C-Methyladenosine Is a Potent Inhibitor of In Vitro Zika Virus Replication and Delays Disease Progression in a Robust Mouse Infection M odel†. The paper is intended to highlight the key problem of the research and approach used to address it.After the paper will conduct a SWOT analysis of the approach suggested by the study to address the research question. Summary of the research in the primary article This article discusses the causes and characteristics of ZIKA viral disease. The main focus of the article is on the identification of ZIKV inhibitors and demonstration of the efficiency of the viral polymerase inhibitorShow MoreRelatedHiv / Aids : A New Form Of Viral Disease1287 Words   |  6 Pagesand governments were struggling to alleviate and fight against HIV/AIDS there emerged a new form of viral disease that terrified the entire population of the planet. ZIKA virus is an emerging killer disease that has symptoms similar to a very mild dengue fever. This paper therefore is a review of an article entitled â€Å"The Viral Polymerase Inhibitor 7-Deaza-2’-C-Methyladenosine Is a Potent Inhibitor of In Vitro Zika Virus Replication and Delays Disease Progression in a Robust Mouse Infection Model†